As one of the important glands in human body, the thyroid hormone secreted by the thyroid gland is very important to the body. If the thyroid function is in an excited state or a passive state, it will directly lead to too much or too little thyroid hormone, and various symptoms of the human body can be reflected immediately. It is necessary to restore the normal function of the thyroid gland through drugs and other means under the guidance of endocrinologists, so that the human body can live a healthy life.

Hyperthyroidism is a series of diseases with high excitability and hypermetabolism caused by excessive secretion of thyroid hormone into the blood circulation. The general clinical manifestations are mainly overeating, emaciation, fear of heat, sweating, excitability and exophthalmos, but if it is not treated in time, it will induce cerebral infarction for a long time. In the past, patients were easily missed or misdiagnosed because of atypical clinical manifestations, but with more and more reports of hyperthyroidism complicated with cerebral infarction in recent years, clinicians have paid more and more attention to it.

The causes of cerebral infarction induced by hyperthyroidism may be related to the following.

1. is related to autoimmune abnormality.

Hyperthyroidism is also one of autoimmune diseases. Because of the abnormal immune function, inhibitory factors and anticoagulant factors are produced in the body. These factors can inhibit the production of prothrombin activation complex, and affect prostacyclin and vascular endothelial cells, causing vasoconstriction and vascular endothelial damage, which can lead to cerebrovascular ischemia or cerebral infarction.

2. It is related to the increase of metabolic rate.

Due to hyperthyroidism, the metabolism of the body is increased, protein, fat and sugar are disordered, the decomposition is accelerated, the permeability of the cell membrane is increased and the cell is deformed, the platelet function is also enhanced, and the adhesion is increased, which will lead to slow blood flow, insufficient cerebral blood perfusion and cerebral infarction.

3. It is related to thrombosis induced by atrial fibrillation.

Thyroid hormone can increase the heart rate, change the heart valve and myocardium, and cause arrhythmia and even atrial fibrillation. Severe atrial fibrillation can lead to thromboembolism, and it can lead to cerebral infarction when it occurs in cerebral vessels. In addition, some studies have found that the secretion of atrial natriuretic peptide in patients with hyperthyroidism is reduced, which leads to a decrease in cardiac output and insufficient arterial blood volume, and also leads to cerebral ischemia and hypoxia.

4. Related to thyroxine toxicity

Due to the excessive secretion of thyroid hormone, patients have increased sympathetic nerve excitability, which leads to the increase of norepinephrine, a vasoconstrictor in the blood, causing vasoconstriction and rising blood pressure. Therefore, some patients with hyperthyroidism will be complicated with hypertension, which is also a high risk factor for inducing cerebral infarction.

Therefore, if you have hyperthyroidism, don't underestimate it. In severe cases, it can induce a stroke and may even be life-threatening. At present, there are three main treatment methods for hyperthyroidism: drugs, iodine 13 1 radiotherapy and surgery. The guidelines recommend drug treatment as the first choice. Most patients take drugs on time and in full course of treatment, and hyperthyroidism can be well controlled. Surgery or iodine 13 1 radiotherapy are only considered for patients who relapse after drug treatment, are allergic to hyperthyroidism drugs or have serious adverse reactions, because the latter two treatments may lead to the risk of patients changing from hyperthyroidism to hypothyroidism, and once they change to hypothyroidism, patients need to take drugs for life.