Vegetables that gout patients should not eat: spinach, lentils, soybeans, peas, seaweed, mushrooms, asparagus, etc.

Vegetables that patients with gout can eat: celery, leeks, wax gourd, pumpkin, cucumber, gourd, amaranth, mustard root, chrysanthemum, celery, cabbage, fungus, mustard, pepper, cabbage, eggplant, etc.

Vegetables that gout patients can eat appropriately: mushrooms, cauliflower, yams, kelp, golden needles, white fungus, etc.

1. The purine content of spinach is: 13.3 mg/100g of spinach.

Purine is an organic compound with the molecular formula C5H4N4. It is a colorless crystal. In the human body, purine is oxidized and turns into uric acid. Excessive uric acid in the human body can cause gout. Seafood and animal meat have relatively high purine content. Therefore, in addition to taking drug treatment (drugs for gout are generally harmful to the kidneys), it is more important for patients with gout to pay attention to dietary taboos.

2. Spinach (Spinacia oleracea L.), also known as Persian, red root, parrot, etc., belongs to the genus Spinach of the Chenopodiaceae family and is an annual herb. The plant can reach a height of 1 meter, with conical roots, reddish, or less white, and halberd-shaped to oval leaves, bright green, with entire edges or a few tooth-like lobes. There are many types of spinach, which can be divided into two varieties: thorny and thornless based on seed shape.

Extended information:

1. According to the purine content in food, we can divide food into low-purine foods (less than 25 mg of purine per 100 grams of food) and medium-purine foods. (containing 25-150 mg of purine per 100 g of food) and high-purine foods (containing 150-1000 mg of purine per 100 g of food).

2. Purine content level: if it exceeds 150 mg/100g, it should not be used; if it exceeds 50-150 mg/100g, it should not be used in the acute stage

; if it is less than 50 mg/100g, it should not be used Suitable for selection. Most vegetables are low in purine, including spinach.

3. Purine diseases: In the process of synthesis and decomposition of purine, a variety of enzymes are involved. Due to congenital abnormalities of enzymes or some unidentified factors, metabolism is disordered, resulting in the synthesis of uric acid. Increase or decrease in excretion can result in hyperuricemia. When the blood uric acid concentration is too high, uric acid is deposited in the joints, soft tissues, cartilage and kidneys in the form of sodium salts, causing foreign body inflammatory reactions in the tissues and becoming the bane of gout. Gout can cause joint swelling.