Can hemolytic jaundice be cured? Can hemolytic jaundice be cured? Hemolytic jaundice is very harmful to newborn babies. When hemolytic jaundice occurs, the baby not only has jaundice in the skin and sclera. And the nerve cells in the brain nucleus may also have jaundice, that is, the formation of nuclear jaundice, which may lead to lifelong paralysis of the baby, affect the baby's intellectual development, and cause hearing impairment. Therefore, it is urgent to treat infants with jaundice.
How to treat hemolytic jaundice? First of all, it depends on whether the baby's jaundice is very serious. Newborn infants with mild jaundice can be irradiated under blue fluorescent lamp to accelerate the oxidation of indirect bilirubin, so as to achieve the effect of yellowing or cooperate with drug treatment. For severe infants, blood exchange and transfusion should be taken as soon as possible, and blood with appropriate blood type should be used instead of blood with antibodies. The earlier the exchange of blood, the better, which can reduce hemolysis and prevent the occurrence of nuclear jaundice. In particular, newborns with severe anemia need blood transfusion.
What I want to remind my mother here is that it is best to breastfeed after the condition is controlled stably to prevent jaundice from getting worse. In addition, jaundice is accompanied by fever, refusal to milk, poor mental health, lethargy, dull eyes and other symptoms. Parents need to carefully observe the baby's state and the change of jaundice. In case of special circumstances, you should be sent to the hospital in time to seek the help of a doctor.
The cause of neonatal jaundice is 1, and bilirubin production is high.
(1) More red blood cells are destroyed: the fetal baby is in an anoxic environment in the mother's womb, and the red blood cells are compensatory increased, but the life span is short, the blood oxygen content increases after birth, and excessive red blood cells are quickly destroyed.
(2) The content of heme oxygenase is high: the content is high within 7 days after birth, and it has great potential to produce bilirubin.
2. Immature liver function
(1) Poor absorption of bilirubin by the liver: the content of Y and Z proteins in hepatocytes is insufficient, resulting in insufficient absorption of bilirubin by the liver.
(2) The function of liver-bound bilirubin is poor: the content of glucuronosyltransferase in the liver is low and its activity is insufficient, so the function of forming bound bilirubin is poor.
(3) Poor function of liver in excreting bilirubin: Poor function of liver in excreting bilirubin easily leads to cholestasis.
3. Intestinal-hepatic circulation characteristics: The normal flora in the intestine has not been established when the newborn is born, and bilirubin entering the intestine cannot be converted into urinary urobilinogen (fecal bile).
Due to the above characteristics, the ability of newborns to ingest, combine and excrete bilirubin is obviously not as good as that of adults, and bilirubin is produced more and excreted less, which is prone to jaundice. Especially when the baby is in a state of hypoxia, meconium discharge is delayed, feeding is delayed, vomiting, dehydration, acidosis, skull hematoma and so on. , will aggravate jaundice.
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