The basis of gout is hyperuricemia, which is more than 420umol/L in men and more than 360umol/L in women (fasting test on different days can be diagnosed if it exceeds the above value twice). Gout is mainly the pain of the first toe joint, and tophi will appear seriously in the later stage. There are three basic treatments for gout: 1. Exercise more. Drink more water. 3. Low purine diet. (In fact, a low purine diet can only reduce uric acid by 20%, with one-third of uric acid excreted from the digestive tract and two-thirds from the urinary system).

Therefore, to treat gout, it is necessary to treat it in the acute attack and remission period of gout.

Acute attack is mainly aimed at pain and anti-inflammatory treatment. In the meantime, it is forbidden to use drugs that reduce uric acid, otherwise it will further aggravate gout attacks. Generally, control inflammation and analgesia regularly for about 3 months (colchicine+non-steroidal drugs are the main ones), and consult the local hospital for specific plans and drugs;

Secondly, you should know whether your constitution is reduced uric acid excretion or excessive uric acid production. The simple method is to measure the 24-hour uric acid urine excretion in the local 3A general hospital. Under normal diet, the excretion of uric acid in 24 hours is more than 800umol/L, and less than 800 is too little. If you are on a low purine diet, the value is adjusted to 600 μm ol/L, as above.

According to the above types, reasonable gout drug treatment is selected, but it is also carried out after 3 months of routine anti-inflammatory and analgesic. Generally, the routine in the market is: inhibiting uric acid synthesis (dominant uric acid synthesis type): allopurinol and febuxostat. . It is Benbromarone tablets that promote uric acid excretion (resulting in too little uric acid excretion), according to local hospital consultation.

The ultimate goal of gout treatment is to relieve joint pain. If gout attacks twice or more in 1 year, it is suggested that the uric acid value should be controlled above 300umol/L and 1 80 umol/l. If 1 year has only1attack or no attack, only uric acid should be controlled below 360 umol/l./kloc-. At the same time, perennial hyperuricemia and gout will lead to the risk of gouty nephropathy, and