Carambola may be a drug for patients with renal failure, but it has not attracted widespread attention because of its narrow influence on people. In recent years, domestic and foreign literatures have reported cases of carambola poisoning in patients with chronic kidney disease. Generally, carambola poisoning can cause intractable hiccups, numbness of limbs, decreased muscle strength, abnormal skin sensation, insomnia, excitement, thinking disorder, seizures, lethargy, coma, diarrhea, hematuria and other poisoning symptoms.
Many doctors' cases also showed that eating carambola led to the aggravation of kidney diseases. For example, primary nephrotic syndrome is prone to relapse after eating carambola, uremic patients on peritoneal dialysis and hemodialysis are prone to poisoning and coma after eating carambola, and even some children have hematuria symptoms after eating carambola. Through experimental research, this may be related to the irritating neurotoxin contained in carambola. In addition, animal experiments also show that carambola can cause glomerular capillary basement membrane damage, epithelial cell foot process damage, leading to hematuria, and its mechanism is probably related to allergic reaction. We also found that the symptoms of poisoning disappeared rapidly after the coma patients were rescued by hemodialysis, suggesting that hemodialysis has a good effect on removing carambola toxin.