How to detect rickets rickets, commonly known as calcium deficiency, is more common in infancy. The main reason is the lack of vitamin d in the body This is a kind of bone matrix calcification disorder, which will cause calcium and phosphorus metabolism disorder in the body and lead to poor bone calcification. Insufficient ultraviolet radiation, insufficient or improper calcium and phosphorus content in food, insufficient demand for vitamin D for rapid growth, chronic respiratory infection, chronic diarrhea, liver and kidney diseases and other chronic diseases that affect calcium and phosphorus absorption are also the causes of rickets in infants.
Due to the slow onset of rickets, it is generally difficult to find it in time and it is not easy to attract attention. The main feature of rickets is that the growing metaphyseal cartilage plate and bone tissue are not completely calcified, and the lack of vitamin D makes the calcification of mature bones incomplete. Rickets is not contagious, but if infants suffer from rickets, their own immunity will be reduced, and they are prone to pneumonia, diarrhea and other diseases, which will affect their growth and development. Therefore, the importance of active prevention and treatment of rickets is particularly prominent.
Rickets, as a common disease, has a great influence on people's health and life. Some patients get worse because they don't know the examination method of rickets, and it is difficult to cure them in the end. Therefore, it is necessary to know more about the examination of rickets in order to receive better treatment. Let's talk about the inspection method of baby rickets first.
1, x-ray examination.
X-ray changes of long bones with rapid skeletal development, especially the distal end of ulna and radius and the proximal end of tibia and fibula, are obvious. This kind of rickets examination is more effective.
2. Laboratory examination: This also belongs to the examination of rickets.
(1) Alkaline phosphatase rises earlier in the course of rickets, but recovers at the latest, which is helpful for examination and diagnosis.
(2) The serum level of 25(OH)D3 or 1, 25 (OH) 2d3 is almost zero in typical rickets, and it is obviously decreased in subclinical rickets, but it can be obviously increased after vitamin D treatment, which is a sensitive and reliable biochemical index.
Treatment of rickets The prone population of rickets is mostly infants. After 3 months, the chances of babies suffering from rickets increased. Therefore, the prevention and treatment of rickets is very important. The treatment of rickets is mainly to supplement vitamin D and calcium to prevent skeletal deformity and recurrence. The treatment of rickets is mainly determined according to the period when children with rickets do not use it, which is divided into active period and recovery period.
1, active rickets: active treatment should be given according to clinical manifestations, aiming at controlling disease activity and preventing deformity.
(1) Mild rickets in active stage: vitamin D 200-300000 IU, oral or intramuscular injection 1 time, interval 1 month, 1 ~ 2 times, calcium agent, 0.5- 1 g each time, daily.
(2) Moderate and severe rickets in active stage: vitamin D is 200,000-300,000 IU, orally or intramuscularly injected 1 time, with an interval of 1 month, which can be given 2-3 times, and calcium agent is 0.5- 1g each time, 2-3 times a day, for 2-3 months.
2, rickets recovery period: for infants and young children in the recovery period of rickets, generally do not need vitamin D, you can spend more sun and improve nutrition. However, in winter and spring, in order to prevent recurrence, 200,000 ~ 300,000 IU of vitamin D can be given once orally or intramuscularly. The above-mentioned therapeutic dose of vitamin D can be maintained for 2-3 months, and it is unnecessary to maintain the dose orally, so as to prevent vitamin D poisoning and get more sunshine.
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